The Biological Mechanism of Turbo-Cancers
Since the first vaccination campaigns in 2021, scientists have been sounding the alarm. Among them, Jean-Marc Sabatier, PhD in cell biology and microbiology and research director at the CNRS, has documented the potential link between mRNA vaccines and the development of rapidly progressing cancers, known as turbo-cancers.
According to his explanations, this phenomenon is due to the overactivation of the AT1R receptor of the renin-angiotensin system. This system plays a central role in cell differentiation and multiplication. The spike protein produced after vaccination binds to the ACE2 receptor, disrupting the degradation of angiotensin 2. In excess, the latter overstimulates the AT1R receptor, which has pro-tumor, pro-angiogenic, and pro-inflammatory properties.
The AT1R receptor also promotes oxidative stress by releasing free radicals that are harmful to cells. Conversely, antagonists of this receptor have demonstrated antitumor activity against several types of cancer, including breast, prostate, and lung cancer. Jean-Marc Sabatier had already described the pro-cancerous activity of disruption of this system via the spike protein of the SARS-CoV-2 virus as early as March 2020, well before the first cases were reported.
